Titlebook: Aging Mechanisms; Longevity, Metabolis Nozomu Mori,Inhee Mook-Jung Book 2015 Springer Japan 2015 Aging.Brain.Longevity.Metabolism.Neurodege

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Conducting Polymer Nanostructures,research. Although most of centenarians remain independent in daily living until over 90s, about 97 % of them contracted chronic diseases including hypertension (63.6 %) and bone fracture (46.4 %). The prevalence of diabetes mellitus (DM) and carotid atherosclerotic plaque were peculiarities of cent

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https://doi.org/10.1007/978-3-540-69323-9patients. Further, many elderly patients are waiting for new therapies for treatment of intractable age-related diseases such as Alzheimer’s disease, Parkinson’s disease, liver cirrhosis, lung failure and renal failure. The development of induced pluripotent stem cell (iPSC) technology opens the doo

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BOOR

https://doi.org/10.1007/978-981-287-038-4 budding yeast, have contributed to a better understanding of the complexity of aging and longevity. Budding yeast gradually loses the ability of producing daughter cells in rich media, and then loses viability in exhausted media during the aging process. According to these distinguishable losses, t

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John A. Wilkinson BSc, MCh, FRCS . have identified many genes and pathways that regulate aging, several of which are conserved in other species, including mammals. In this chapter, we describe longevity-regulatory pathways including insulin/IGF-1 (insulin-like growth factor 1) signaling, TOR (target of rapamycin) signaling, autoph

BRIEF

Deformities of the anterior thoracic wall,ases have become the major threats to human health and serious social burdens worldwide. Recently, research in neurodegenerative diseases has entered the Renaissance age, as several genes responsible for the diseases were identified. In this chapter, we explain why . (.) is an excellent animal model

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