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Titlebook: Advances in Research on Neurodegeneration; Volume 10 R. Horowski,Y. Mizuno,M. B. H. Youdim Conference proceedings 2003 Springer-Verlag Wien

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Introduction to Numerical Methods, been suggested that .-DOPA itself may contribute to the progression of Parkinson’s disease. This hypothesis is, for many clinicians, the rationale for postponing the employment of and reducing the applied dosage of .-DOPA and for beginning therapy with dopamine receptor agonists or the monoamine ox
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Solution of the Navier-Stokes Equations,s, both in vitro and in vivo. Specifically, GDF-15 promotes survival and differentiation of embryonic rat dopaminergic neurons, but not of other neuron populations, with the exception of serotonergic raphe neurons. The neurotrophic effect of GDF-15 seems to be direct and not mediated through glial c
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Turbulent-Flow Models and Calculationss attached to gelatin microcarriers (Spheramine®) in rodent and non-human primate models of Parkinson’s disease (PD) produces long term amelioration of motor and behavioral deficits, with histological and PET evidence of cell survival without immunosuppression. Long-term safety in cynomologous monke
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Advances in Research on Neurodegeneration978-3-7091-0643-3Series ISSN 0303-6995
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0303-6995 Overview: 978-3-211-83907-2978-3-7091-0643-3Series ISSN 0303-6995
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Possible role of intramembrane receptor-receptor interactions in memory and learning via formation l a molecular mechanism that can contribute to change synaptic weights may be represented by multiple interactions between membrane receptors forming aggregates (receptor mosaics) via oligomerization at both pre- and post-synaptic level..These assemblies of receptors together with inter alia single
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