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Titlebook: Advances in Myocardiology; Peter Harris,Philip A. Poole-Wilson Book 1985 Springer Science+Business Media New York 1985 cardiology.electrop

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https://doi.org/10.1007/978-981-15-0497-6easier technically and interpretatively to carry out experiments in vitro, much of the work described here is based on this approach. It is important, however, to attempt to relate phenomena observed in vitro to the in vivo situation. This can be done by attempting to simulate in vivo conditions (e.
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Treatment of Rheumatic Diseases,ast, the primary metabolite of ethanol, acetaldehyde, markedly diminishes synthesis despite its chronotropic and inotropic effects. On the other hand, after 11–13 weeks of ethanol-drinking during growth and maturation, the synthetic capacity of the working right ventricle was decreased when measured
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Heat Transport System Thermal Hydraulics,vation of perfusion pressure from 60 to 120 mm Hg increased oxygen consumption in control–beating but not in arrested–drained preparations. Energy availability, as assessed by adenylate energy charge or creatine phosphate/creatine ratio, or both, was increased in arrested–drained hearts supplied wit
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Definitions and Basic Principles,n injury. We have hypothesized that activated neutrophils are capable of disrupting myocardial function via an oxygen free-radical mechanism. Human neutrophils activated with phorbol myristate acetate disrupted calcium transport by canine cardiac sarcoplasmic reticulum, and this process was inhibite
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Open Air-Brayton Gas Power Cycle,tes. These reduced-oxygen intermediates include the superoxide anion (·O.), hydrogen peroxide (H.O.), and the hydroxyl radical (·OH), which are highly reactive, short-lived species. Normally, intracellular enzyme systems that include superoxide dismutase, catalase, and glutathione peroxidase are res
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