Titlebook: Advances in Dementia Research; K. Jellinger,R. Schmidt,M. Windisch Conference proceedings 2000 Springer-Verlag Wien 2000 Assessment.Nervou

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https://doi.org/10.1007/978-3-7091-6781-6Assessment; Nervous System; genetics; imaging techniques; lead; neuroimaging; neurons; neurotoxicity; pathop

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978-3-211-83512-8Springer-Verlag Wien 2000

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Conference proceedings 2000, the aetiology and pathogenesis of most dementia disorders are still poorly understood, and early exact diagnosis as a prerequisite of effective treatment needs to be improved. The present volume includes the contributions of renowned experts in the field of neurodegeneration presented at the Inter

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Peter M. Steinert,Dennis R. Roops are markedly upregulated in AD tissue while those of the complement inhibitors are almost unchanged. The upregulations for CRP and CDllb in AD hippocampus are comparable to those in osteoarthritic joints. This lends further support to the hypothesis that chronic inflammation may be causing neuronal death in AD.

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Eugene M. Johnson Jr.,Pamela Toy Manning were determined. This revealed that treatment of the cells with apoE4, the AD risk factor, resulted in a marked increase in the levels of secreted APPs. This effect was dose dependent (ED. ≅ 2.5μg/ml) and isoform specific in that apoE3 had virtually no effect on the secretion of APPs.

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