Titlebook: Adrenergic Mechanisms in Myocardial Ischemia; Gerd Heusch,John Ross Conference proceedings 1991 Springer-Verlag Berlin Heidelberg 1991 arr

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https://doi.org/10.1007/978-3-319-12066-9subendocardium..Physiologic interventions such as exercise can alter myocardial oxygen requirements substantially. Vasodilator reserve will be utilized in those parts of the myocardium that have it available, in order to meet the augmented myocardial flow requirements associated with exercise. In po

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Initial Requirements and Use Cases,ayers during stellate ganglion stimulation. However, contractile function was increased only in subendocardium. It was proposed that only in deeper muscle layers does an α. -coronary constriction impose a flow-limitation on contractile function. Finally, recent results indicate that myocardial ische

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Guided Tour of the Development Process,f the α.-adrenoceptor antagonist rauwolscine (0.2mg/kg i.V.). Administration of rauwolscine, however, did not prevent the reflex increase of left ventricular pressure and regional myocardial oxygen consumption. Comparable increases in poststenotic coronary resistance during BCO were found in dogs wh

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β-Adrenergic Receptors and the Gs Protein in Myocardial Ischemia and Injury978-3-7091-9487-4

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β- and α-Adrenoceptor-Agonists and -Antagonists in Chronic Heart Failure978-3-322-87710-9

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Supersensitivity of the Adenylyl Cyclase System in Acute Myocardial Ischemia: Evaluation of Three In978-3-322-86015-6

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Adrenergic Vasomotion in the Coronary Microcirculation978-3-663-02803-1