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Titlebook: Adenosine and Adenosine Receptors; Michael Williams Book 1990 The Humana Press Inc. 1990 Nervous System.PAC.Purine.adenosine.biochemistry.

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Urologische Erkrankungen im Kindesalterripheral (PNS) and central nervous system (CNS) (for reviews, . Phillis and Wu, 1981; Dunwiddie, 1985). Many investigators now commonly refer to these substances as neurotransmitters or neuromodulators. The use of these terms tends to imply that purines are released from, accumulated by, and have ac
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Wichtige Anomalien der Urogenitalorgane filtration rate, renin secretion, urine flow, sodium excretion, transmitter release from renal efferent nerves, and the activity of renal afferent nerves. Many of these effects are produced by adenosine receptors, since the effects are antagonized by alkylxanthines and mimicked by adenosine analogs
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Harnsteinerkrankung (Urolithiasis)Van Calker, 1985), and differentiated by pharmacological and functional activity as well as differences in regional distribution (Snyder, 1985; Williams, 1987; Jarvis, 1988). Adenosine receptors in brain tissue, like other receptors, occur in the greatest density in this organ as compared to their d
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Harnsteinerkrankung (Urolithiasis)s of the immune and host defense systems communicate through the medium of receptor-ligand interactions. Indeed, many of the receptors that are present on nervous tissue are present on neutrophils, lymphocytes, macrophages, and basophils (Goetzl, 1985).
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Wichtige Anomalien der Urogenitalorganesubject (Baer and Drummond, 1979; Berne et al., 1983; Gerlach and Becker, 1987) and several others of no less importance (Stone, 1985; Stefanovich et al., 1985; Pelleg et al., 1987; Paton, 1988; Ribeiro, 1989; Jacobson et al., 1990). Receptor binding studies have provided preliminary evidence for ne
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Harnsteinerkrankung (Urolithiasis)The effects of adenosine and its related nucleotides on cardiovascular function were initially reported by Drury and Szent-Györgyi (1929), who noted in studies performed in a variety of mammalian species, that these compounds were potent bradycardic and blood pressure lowering agents with marked vasoconstrictor effects in the kidney.
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Harnsteinerkrankung (Urolithiasis)er, the many documented inhibitory effects of adenosine on neurotransmitter release in mammalian tissue coupled with the psychomotor-stimulant effects of adenosine receptor antagonists (alkylxanthines) have led to the suggestion that adenosine mediates an “inhibitory tone” in the CNS (Harms et al., 1978).
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