Titlebook: White Matter Injury in Stroke and CNS Disease; Selva Baltan,S. Thomas Carmichael,John H Zhang Book 2014 Springer Science+Business Media Ne

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White Matter Injury in Global Cerebral Ischemiaiffuse axonal injury after traumatic brain injury, white matter lesions occur not only as a result of secondary degeneration caused by neuronal injuries in the gray matter, but also as a direct consequence of the primary ischemic insults. Not enough attention has been directed to the molecular and c

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White Matter Injury After Experimental Intracerebral Hemorrhagelf of ICH patients die and only 10–20 % return to normal activities of daily living. Although the incidence of spontaneous ICH is estimated at ~10–15 % of all strokes, approximately 2 million patients are affected yearly worldwide. Besides spontaneous ICH, intracerebral bleeds also occur following t

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White Matter Repair in Subcortical StrokeThe temporal changes occurring in white matter stroke such as acute loss of axons and astrocytic and microglia responses are likely to change the milieu of the stroke white matter into an inhibitory environment to the remyelination process. Based on the limited number of studies investigating acute

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White Matter Injury in Subarachnoid Hemorrhage in Humansnisms lead to white matter injury in aSAH: early brain injury, delayed brain injury, and hydrocephalus. The problem of white matter injury after SAH is a relatively understudied topic, and little is known about the specific mechanisms that cause damage.

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Degenerative Brain Diseases and White Matter Injuryn glia and especially oligodendrocytes and their roles in achieving optimal brain function as well as triggering degenerative brain processes. The evidence suggests that recently evolved vulnerable aspects of the human brain such as myelin and the homeostatic repair mechanisms triggered by its break

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Unmyelinated and Myelinated Axons Exhibit Differential Injury and Treatment Responses Following Traustinct from those of myelinated axons. A theoretical framework is presented which predicts an elevated risk to TBI, in the unmyelinated fibers, due to a disproportionately high membrane-to-cytoplasm ratio and extensive axolemmal exposure to aberrant extracellular conditions prevailing after injury.

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White Matter Damage in Multiple Sclerosislination in the white and gray matter of the brain and spinal cord, with features of inflammation, oligodendroglial death, and axonal degeneration. This pathology most commonly affects young adults and leads to significant neurological disability, such as sensory disturbances, lack of motor coordina

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Book 2014ies, neurodegenerative disorders including Alzheimer‘s disease, traumatic brain injuries, carbon monoxide poisoning, and drug or alcohol overdoses. The extent of white matter injury is extremely important to patient outcomes. Several recent technological developments including advanced neuroimaging