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Titlebook: Topics in Pathophysiology of Hypertension; Herman Villarreal (Head),Mohinder P. Sambhi (Profe Book 1984 Martinus Nijhoff Publishers, Bosto

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Cation Transport Abnormalities in Human and Rat Essential HypertensionFrom the pioneering observations of Dahl and associates [1] on the positive correlation between the prevalence of hypertension and salt intake in different populations, several investigators have attempted to find a relationship between abnormal sodium metabolism and essential hypertension.
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Evidence against Raised Intracellular Sodium Causing Myogenic Contraction in Rat Resistance VesselsA number of recent findings have pointed to possible molecular mechanisms that could account for the long-suspected association between sodium intake and the development of essential hypertension.
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Erythrocyte Na+ Handling in Rat Genetic Hypertension. Transmembrane pathways in the red cells of genetically hypertensive rats are also altered [2]. This paper summarizes our previous findings on Na. transport systems in SHR and SbH and reports some new data on the effect of chronic and acute Na. loads on cellular Na. handling.
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The Sodium/Lithium Exchange Mechanism in Essential Hypertension: Is it a Sodium/Proton Exchanger? patients with essential hypertension, there has been considerable interest in this cation exchange system, which can mediate sodium-lithium exchange and sodium-sodium exchange in a strictly coupled 1:1 transmembrane exchange process.
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Presynaptic α-Autoreceptors: Pharmacological Properties and Physiological Function between postganglionic sympathetic neurons and muscle cells [1, 2]. The terms α. and α. were loosely suggested [3], then used to refer to post- and presynaptic α-receptors [4], and finally generalized [5, 6].
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978-94-009-6743-4Martinus Nijhoff Publishers, Boston 1984
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Topics in Pathophysiology of Hypertension978-94-009-6741-0Series ISSN 0166-9842
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