Titlebook: Systems Biology of Tumor Dormancy; Heiko Enderling,Nava Almog,Lynn Hlatky Book 2013 The Editor(s) (if applicable) and The Author(s), under

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Regulation of Tumor Cell Dormancy by Tissue Microenvironments and Autophagyesidual cells. Herein, we review recent evidence in support of genetic and epigenetic mechanisms driving dormancy. We also explore how therapy may cause the onset of dormancy in the surviving fraction of cells after treatment and how autophagy may be a mechanism that maintains the residual cells tha

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Tumor Dormancy, Oncogene Addiction, Cellular Senescence, and Self-Renewal Programs therapy. One important mechanistic insight into tumor dormancy is that oncogene addiction might involve regulation of a decision between self-renewal and cellular senescence. Recent evidence suggests that this decision is regulated by multiple mechanisms that include tumor cell-intrinsic, cell-auto

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Tumor Dormancy and Cancer Stem Cells: Two Sides of the Same Coin?olved in controlling the dormant state of a tumor can also govern CSC behavior, including cell cycle modifications, alteration of angiogenic processes, and modulation of antitumor immune responses. In fact, quiescence and immune escape are emerging hallmark features of at least some CSCs, indicating

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drink in the world. More than half of the world’s population is habituated of tea drinking. India, Sri Lanka, Indonesia, China and Japan are the traditional tea producers in the world. The demand for tea is ever increasing, and more and more land is being brought under tea plantations. Tea is curren

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