Titlebook: Symposium on Pathology of Axons and Axonal Flow; Organized by the Öst Reinhard L. Friede,Franz Seitelberger Conference proceedings 1971 Spr

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大火

贪婪的人

,Fine Structural Changes of Neurites in Alzheimer’s Disease,, floccular dense bodies; 2. interlacing bundles of twisted tubules; 3. aggregates of mitochondria usually associated with membranous dense bodies; 4. arrays of irregular vesicular profiles, and 5. granular and vacuolar disintegration. A relationship between amyloid deposits and abnormal neurites in

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Mitochondrial Changes in Axonal Dystrophy Produced by Vitamin E Deficiency,d on a vitamin E deficient diet. The alterations include enlargement accompanied by pleomorphism, swelling, derangement in the structure of cristae and two types of intramitochondrial deposits. Many of these changes are also observed in other types of cell injury and therefore cannot be considered a

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伟大

松果

Employee

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Slow and Rapid Transport of Protein to Nerve Endings in Mouse Brain,oteins, which includes microtubular protein, are transported relatively slowly. In contrast, fucosyl glycoproteins are representative of a group of proteins which are transported to nerve endings at a more rapid, but measurable rate. Evidence is also presented for local incorporation of glucosamine