Titlebook: Steroid Hormones and the T-Cell Cytokine Profile; G. A. W. Rook,S. Lightman Book 1997 Springer-Verlag London Limited 1997 asthma.autoimmun

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Immunological consequences of inhibiting dehydroepiandrosterone (DHEA) sulfatase in vivo,n of antigen is usually a very efficient process, requiring the integrated activity of a variety of cell types. It is the recruitment and activation of these cells that leads to an appropriate immune response. Indeed an inappropriate immune response can often lead to pathology such as that seen in a

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Glucocorticoid regulation of , in host resistance to mycobacteria,the adrenal cortex is stimulated primarily by adrenocorticotrophin (ACTH), which in turn, is controlled by Corticotrophin-releasing hormone (CRH) derived from the hypothalamus [1]. Circadian rhythms or “episodic” increases and decreases occur during each day. Stress overrides feedback regulation of

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Human and murine tuberculosis as models for immuno-endocrine interactions,losis as a model on which to base discussion of the ways in which these changes, even when not disease-specific, may impact upon the function of T lymphocytes during chronic inflammation. Other chapters in this volume amplify further several of the topics highlighted in this one.

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Corticosterone and the hypothalamic-pituitary-adrenal (HPA) axis in autoimmune diseases,ne system. As many of the biological parameters are susceptible to genetic variation, it is not surprising that inbred strains of laboratory animals differ strikingly in the way that they respond to various antigens. The differences concern two aspects of immunity.

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Overview: 978-1-4471-1238-9978-1-4471-0931-0