Titlebook: Software Language Engineering; 7th International Co Benoît Combemale,David J. Pearce,Jurgen J. Vinju Conference proceedings 2014 Springer I

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Petra Kaufmann,Martin Kronegger,Andreas Pfandler,Martina Seidl,Magdalena Widlar mechanisms for the alteration of synaptic function are not completely understood. We conducted a series of studies to investigate the molecular and morphological mechanisms underlying these synaptic alterations in the hippocampus. Rat brains were subjected to 15 min ischemia followed by 0 min, 30

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Markus Voelter,Janet Siegmund,Thorsten Berger,Bernd Kolbry, the regional expression of Bcl-w protein in rat brain was examined at 6-72 h after 90 min transient middle cerebral artery occlusion. Bcl-w protein, while constitutively expressed at low levels in non-ischemic brain, was found to be overexpressed in ischemic brain at all time points studied. Up

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Andrei Chiş,Tudor Gîrba,Oscar Nierstraszcular compartment into the extravascular compartment, and disturbance of cell-cell relationships within the extravascular compartment, proteases identified in the target tissue following the ischemic insult which lesion major alterations in tissue barriers occur. These involve disruption of the cere

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Thomas Kühn,Max Leuthäuser,Sebastian Götz,Christoph Seidl,Uwe Aßmanncular compartment into the extravascular compartment, and disturbance of cell-cell relationships within the extravascular compartment, proteases identified in the target tissue following the ischemic insult which lesion major alterations in tissue barriers occur. These involve disruption of the cere

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Mila Dalla Preda,Saverio Giallorenzo,Ivan Lanese,Jacopo Mauro,Maurizio Gabbriellimic insult. The delayed neuronal death of CAl pyramidal cells of the hippocampus is a classic example. The report of the phenomenon boosted research in the field, as it became evi­ dent that ischemic damage is not a sudden event but a process potentially suscep­ tible to therapeutic intervention. In