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Titlebook: Signal Transduction and the Gasotransmitters; NO, CO, and H2S in B Rui Wang Book 2004 Springer Science+Business Media New York 2004 ATP.Cal

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Chemical Interaction of Nitric Oxide With Protein Thiolsaling is a rapidly emerging discipline that is relevant to nearly every field of medicie. However, the field remains hampered by imprecise assays and biochemical controversies. This chapter reviews the bioactivities and metabolism of physiological SNO compounds as well as the interaction of these compounds with aasotransmitters.
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Nitric Oxide and Voltage-Gated Ca2+ Channelsene expression, cell proliferation, and apoptosis. This chapter reviews numerous studies highlighting direct and indirect modulatory effects of NO on various types of voltage-gated Ca. channels and discusses the functional implications of the interaction of NO with voltage-gated Ca. channels.
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Carbon Monoxide and Signal Transduction Pathwaysare becoming the focus of intense investigations. This chapter reviews the known signal transduction pathways of CO with a special emphasis on the roles of guanylate cyclase, the mitogen-activated protein kinases, and nuclear factor-κB.
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The Evolution of Gasotransmitter Biology and Medicinelth effects. Among pollutant gases are nitric oxide (NO), carbon monoxide (CO), and hydrogen sulfide (H.S). Over several decades, studies from numerous laboratories have demonstrated that gases such as NO, CO, and H.S not only are generated in the human body but also play important physiological rol
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Nitric Oxide most if not all cell types, including neuronal tissue. NO plays important functions as a signaling substance in mammalian and nonmammalian species. Despite the simple nature of the molecule, the chemistry of NO and its adjuncts have proved to be quite complex and its cellular actions are now known
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Chemical Interaction of Nitric Oxide With Protein Thiolsecificity and to prevent nitrosative stress. This regulation is achieved through metabolic control of S-nitrosothiol (SNO) synthesis and catabolism, as well as through cellular localization. Conditions involving excessive SNO accumulation (i.e., nitrosative stress) and regional SNO depletion (i.e.,
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