Titlebook: Sensory Neuropathies; Arthur K. Asbury,Herbert Budka,Elfriede Sluga (em. Conference proceedings 1995 Springer-Verlag/Wien 1995 AIDS.Nervou

栏杆

plasma

Pathomorphology of hereditary sensory neuropathiesedicated on clinical differences. This classification of HSAN I–V seems to be uncontested, at the present time. Morphologically, individual forms I–V only differ in the non-specific loss or lack of myelinated and unmyelinated nerves in varying degrees in that in HSAN II large myelinated axons are mo

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发炎

Spectrum of acquired sensory neuropathy: Clinical, electrodiagnostic, and pathologic studiesredominance (76%). Characteristically the neuropathy was asymmetric at onset (73%), evolved in a subacute (45%) or chronic fashion (40%), and started with upper extremity pain (67%). Ataxia (52%) and Adie’s pupils (18%) also seemed characteristic of this neuropathy. In 16 patients (48%) underlying c

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Ataxic neuropathieshysiologic findings. Individuals with normal sensory nerve action potentials (SNAP’s) can be evaluated for dorsal radiculopathies, myelopathies masquerading as neuropathies, or hysteria. Findings indicative of demyelination are seen in some rare cases of acute and chronic inflammatory demyelinating

indicate

Is acute ataxic neuropathy a distinct entity? Clinical-electrophysiological and morphological study or significant motor weakness, distinct electrophysiological abnormalities, frequently persisting and disabling ataxia distinguish this rare disorder from the more benign Miller Fisher syndrome and more commonly observed G. B. Syndrome with residual ataxia.

项目

Drug induced sensory neuropathiesensory neuropathies (SN) cytostatics are at the top, followed by very differently acting and used substances like for instance almitrine, a bronchodilatator, the antiretroviral agent dideoxycytidine (dd) or vitamin B 6-abuse (Fig. 1). The only chiefly motor neuropathy among the cytostatics is caused

很是迷惑

idiopathic

Peripheral nerve dysfunction in chronic obstructive pulmonary diseasel nerve dysfunction. None of the COPD patients had any obvious cause of peripheral neuropathy. Diagnosis of definite peripheral neuropathy clinical and subclinical — has been done in 27% of the hypoxemic COPD patients vs 9% of the normoxemic COPD patients..The mean values of the median and peroneal

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Conference proceedings 1995s and physicians, pure sensory neuropathies and neuro­ nopathies have received comparably less attention, despite of the consider­ able morbidity they may cause in the individual patient. This prompted us to organize an International Symposium on Sensory Neuropathies which was held in Vienna, Septem