Titlebook: Neuronal Nicotinic Receptors; Francesco Clementi,Diego Fornasari,Cecilia Gotti Book 2000 Springer-Verlag Berlin Heildelberg 2000 Alzheimer

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L. G. Sivilotti,D. Colquhoun,N. S. Millarnology. 2. What are the hot research topics/interests in the technology? 3. What kind of mistakes/assumption that people has tried, so to avoid repeating the mistakes. 4. What can be the special interests/probl978-1-4899-9867-5978-0-387-73819-2

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Gene Structure and Transcriptional Regulation of the Neuronal Nicotinic Acetylcholine Receptorso. Pools were scored as positive if the mRNAs they bound drove the synthesis of immunoprecipitable protein. They were divided up until single clones capable of retaining receptor mRNA were isolated (BALLIVET et al. 1982). In short order, groups led by Barnard, Changeux, Heinemann, and Numa reported

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Transcriptional Regulation of Neuronal nAChR Subunit Geneslevel of the subunits themselves. For example, particular subunits are obligatory for agonist binding and therefore these are required in all functional subtypes. Additionally, selective subunit-subunit interactions allow for only particular combinations of subunits present within a cell to assemble

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The Structures of Neuronal Nicotinic ReceptorshR subunit and the remainder of a 5-HT. receptor subunit and produce a receptor with the channel properties of a 5-HT. receptor which is activated by ACh (EISELE et al. 1993). Actually, there are significant sequence differences between the subunits of the receptors in the superfamily, so in general

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Neuronal Nicotinic Acetylcholine Receptors: From Biophysical Properties to Human DiseasesD- aspartate, NMDA), the family of the purinergic receptors, and the family of the nicotinic acetylcholine receptors (nAChRs). This last family includes the 7-aminobutyric acid (GABA)-A receptors, the glycine receptors, the serotoninergic 5-hydroxytryptophan (5-HT.) receptors, and the nAChRs (ORTELL

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