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Titlebook: Nephrocalcinosis Calcium Antagonists and Kidney; Karl-Horst Bichler (Medical Director),Walter Ludwi Book 1988 Springer-Verlag Berlin Heide

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Factors Influencing Renal Calcium Excretion toxication and the milk alkalii syndrome. In all of these disorders there are potential derangements of factors that normally regulate renal tubular calcium reabsorption. Thus, it is important to gain understanding of the possible reasons for nephrocalcinosis and these other disorders by reviewing
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Ca2+ Antagonists — Mode of Action and Pharmacodynamicsrm Ca. antagonist (or Ca. entry blocker or slow channel blocker) is being used for a whole group of substances which basically have the same mode of action despite their heterogenous chemical structure [31]. The group of Ca. antagonists comprises
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Pathological Anatomy, Etiology, and Pathogenesis of Nephrocalcinosisf view; only a few fundamental light and electron microscopic studies are available (Randall and Melvin 1937; Jaccottet 1957; Caulfield and Schrag 1964; Anderson 1982), but the physiology and pathophysiology of the renal handling of calcium have evoked increasing interest.
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Nephrocalcinosis in the Kidney of the Rat on Atherogenic Diet and the Effect of Calcium Antagonists the localization of concrements found, indicating that the pathogenesis is to a large extent unknown and that renal lesions present themselves in several ways. Probably these lesions are partly dystrophic calcifications caused by cell damage, as they are known to occur in other organs [6]; for exam
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The Unique Chemical Composition of Nephrocalcinosis in Experimental Renal Insufficiency, Disturbancen of the soft tissues. These entities were known to ancient physicians and the Hippocrates recommendation to avoid large quantities of milk for an infant’s nursing (some should be substituted with a diluted wine) represents an indication of a deep intuitive awareness of what Sir Hamphry Davy will re
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Effect of Calcium Antagonist Diltiazem on Acute Renal Failure: Experiments on Animals and Clinical Sogic processes [2]. Apart from its role in the pathogenesis of arterial hypertension the contribution made by abnormal calcium homeostasis to cellular damage is more and more clearly appreciated [3]. It should be remembered that the resting extracellular calcium concentration is greater than the int
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