Titlebook: Maturation Phenomenon in Cerebral Ischemia III; Defensive Mechanisms Umeo Ito,Cesare Fieschi,Igor Klatzo Conference proceedings 1999 Spring

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The Role of Caspase-3-like Protease in the Hippocampus After Transient Global lschemia member of the interleukin-converting enzyme (ICE) protease family. At both message and protein levels, this gene’s product was upregulated in selectively vulnerable regions following ischemia. Coincident with cell death, the precursor protein was proteolytically cleaved and the protease activity in

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Studies of Neuronal Necrosis and Apoptosis after Global Cerebral Ischemia in Superoxide Dismutase Troxide-radical-mediated brain injury, including focal transient ischemia/reperfusion and excitotoxic neuronal injury in culture. The development of mice that overexpress and underexpress the CuZnSOD-deficient gene (.) has provided a model for assessing the role of CuZnSOD in ischemic brain injury. Em

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Apoptosis-Related Genes Are Expressed in the Rat Model of Subarachnoid Hemorrhage the brain and detected expression of apoptosis-related genes in the hippocampus. Intracranial carotid bifurcation of the rat was perforated by a 3–0 nylon and subarachnoid hemorrhage induced. The intracranial pressure rose immediately to a level similar to systemic arterial pressure for 30 s to 1 m

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Stimulation of β2-Adrenoceptors Induces Nerve Growth Factor and Inhibits Apoptosis in Rat Brain Aftereased expression of nerve growth factor (NGF), since the neuroprotective effect of clenbuterol, a β.-adrenergic drug, was abolished, or at least reduced, by a β-blocker, NGF antibodies, p75 antibodies and NGF antisense oligodeoxynucleotide (ODN). Clenbuterol also reduced the infarct volume after oc

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