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Titlebook: Mathematical Methods of Environmental Risk Modeling; Douglas J. Crawford-Brown Book 2001 Springer Science+Business Media Dordrecht 2001 ca

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书目名称Mathematical Methods of Environmental Risk Modeling
编辑Douglas J. Crawford-Brown
视频video
图书封面Titlebook: Mathematical Methods of Environmental Risk Modeling;  Douglas J. Crawford-Brown Book 2001 Springer Science+Business Media Dordrecht 2001 ca
描述.Mathematical Methods of Environmental Risk Modeling.provides a working introduction to both the general mathematicalmethods and specific models used for human health risk assessment.Rather than being purely an applied math book, this book focuses onmethods and models that students and professionals are likely toencounter in practice. Examples are given from exposure assessment,pharmacokinetic modeling, and dose-response modeling.
出版日期Book 2001
关键词calculus; differential equation; model; modeling; numerical methods; statistics; ecotoxicology
版次1
doihttps://doi.org/10.1007/978-1-4757-3271-9
isbn_softcover978-1-4419-4900-4
isbn_ebook978-1-4757-3271-9
copyrightSpringer Science+Business Media Dordrecht 2001
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Douglas J. Crawford-Brownat sympathomimetic agents evoked their effects on different tissues with different orders of potency. . .- and . .-Adrenoceptors differ in their relative affinities for the endogenous catecholamines noradrenaline and adrenaline. At . . .adrenoceptors, which mediate cardiac effects and lipolysis, nor
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Douglas J. Crawford-Brownat sympathomimetic agents evoked their effects on different tissues with different orders of potency. . .- and . .-Adrenoceptors differ in their relative affinities for the endogenous catecholamines noradrenaline and adrenaline. At . . .adrenoceptors, which mediate cardiac effects and lipolysis, nor
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Douglas J. Crawford-Brownat sympathomimetic agents evoked their effects on different tissues with different orders of potency. . .- and . .-Adrenoceptors differ in their relative affinities for the endogenous catecholamines noradrenaline and adrenaline. At . . .adrenoceptors, which mediate cardiac effects and lipolysis, nor
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Douglas J. Crawford-BrownARF is present when arterial oxygen tension (PaO.) is low, the alveolar to arterial oxygen gradient is greater than normal, and/or the arterial carbon dioxide tension (PaCO.) is high in the absence of compensated metabolic alkalosis [1]. In the septic patient, ARF is considered synonymous with adult
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sepsis, trauma, and aspiration of gastric contents [19]. Less frequently occurring situations are amniotic fluid embolization, acute pancreatitis, radiation lung injury, disseminated intravascular coagulation, and ARDS induced by certain drugs [13]. ARDS was described approximately 20 years ago, yet
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