Titlebook: Magen; H. Berndt,H. Bünte,P. F. Ziegler Book 1974Latest edition Springer-Verlag Berlin · Heidelberg 1974 Magen.Magenkrankheit

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W. Frommhold,R. Herzerots of figuresHepatic sinusoidal cells such as sinusoidal endothelial cells, Kupffer cells, hepatic stellate cells, and pit cells play an important role in hemodynamic and metabolic function and are involved in various liver diseases. The frequent involvement of sinusoidal cells in pathologic condit

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). In 1898, however, he misconstrued these cells as phagocytic cells. The original stellate cells were rediscovered in 1971. These cells are located perisinusoidally, and extend several long branching cytoplasmic processes onto the abluminal surface of the endothelial cells. These cytoplasmic proces

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l lamina. Fenestrae measure about 150nm, occur at a frequency of 9–13/mm., and occupy 6%– 8% of the endothelial surface (porosity). These filter characteristics determine the exchange between the blood and the parenchymal cells, and are influencing the transport of lipoproteins including cholesterol

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M. Classen,W. Domschkeent of various forms of liver failure, hepatitis, and acute In recent years, substantial progress has been made in the approaches to diagnosis and treatment of severe liver diseases in the pediatric ICU, from various forms of liver failure and hepatitis to acute and chronic liver disease. New techni

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M. Classen,J. Phillipent of various forms of liver failure, hepatitis, and acute In recent years, substantial progress has been made in the approaches to diagnosis and treatment of severe liver diseases in the pediatric ICU, from various forms of liver failure and hepatitis to acute and chronic liver disease. New techni

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G. Rettenmaierve divergent hepatic outcomes, with those with NAFL having benign course, whereas those with nonalcoholic steatohepatitis (NASH) being at risk for development of cirrhosis, liver failure, and hepatocellular carcinoma. Most patients with NAFLD are asymptomatic and commonly have other metabolic comorb

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K. Krentzspected acute and/or chronic liver disease. While commonly referenced as ., the typical liver panel consists of elements that reflect hepatocellular injury, biliary tract disease, bilirubin metabolism, and protein synthesis. Despite its exclusion in the basic liver panel, an elevation in prothrombin