Titlebook: Lipoprotein(a); Karam Kostner,Gerhard M. Kostner,Peter P. Toth Book 2023 The Editor(s) (if applicable) and The Author(s), under exclusive

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Maya S. Safarova,Patrick M. Moriartyf nuclear proliferation and deterrence. To the contrary, citing the Quran, Khomeini issued a fatwa against weapons of mass destruction on the grounds that they kill innocent civilians..Two interrelated developments, however, compelled the regime to consider a nuclear weapon. The Ayatollah and his Is

Anticoagulant

pester

助记

David Sullivan,Catherine Woolnough,Nimalie Perera,Jay Ramanathan,Tony Badrickhe Anglo-American invasion of Iraq was to “liberate” its people from the tyranny of Saddam Hussein and the despotism of his regime. The second most important reason was to destroy alleged weapons of mass destruction accumulated in Iraq by Saddam Hussein. The third reason we must consider as most imp

nugatory

Noemie Clouet-Foraison,Tomas Vaisar,Santica M. Marcovinacrimes against their own citizens and neighboring countries, including genocide, crimes against humanity and war crimes. Transitional justice was a new concept for the Middle East and for Iraqis, but regime change, occupation and a “new Iraq” had a great impact and pushed the new political elite to

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,60 Years of Lp(a) Research: From Ouchterlonys Double Diffusion to Copy Number Variation and a Signicientist Kare Berg in 1963 until the recent development of drugs lowering Lp(a) for the prevention and treatment of cardiovascular disease are described. Important milestones were the purification and characterization of Lp(a) by Christian Ehnholm and colleagues in Finland, turnover studies elucidat

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Lp(a) Biochemistry, Composition, and Structure,uent studies from numerous laboratories revealed that Lp(a) consists of an LDL-like core particle and the characteristic apo(a) glycoprotein both linked by one disulfide bridge. Since the lipid composition of LDL varies quite considerably among patients, it is evident that this might be also the cas

heterodox

Genetics of Lipoprotein(a),genetic trait with extremely skewed distributions of levels which range from absent to >200 mg/mL in human plasma from healthy individuals. The major gene controlling the quantitative Lp(a) trait is LPA which codes for apo(a). Two major types of variation in the gene determine Lp(a) levels. First, a

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