Titlebook: Left Ventricular Hypertrophy; Physiology versus Pa Ernst E. Wall,Arnoud Laarse,Albert V. G. Bruschke Book 1999 Springer Science+Business Me

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Assessment of Left Ventricular Hypertrophy: A Comparison of Electrocardiography, Echocardiography a used to determine LVH are the electrocardiogram (ECG), the echocardiogram and magnetic resonance imaging (MRI). There are differences in the results obtained by all three techniques as illustrated by the fact that LVH prevalence in the general population increases with age by approximately 1 to 10%

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Hypertrophy and Hypertension,tension by increasing the mass of the left ventricle. As a result, the heart in hypertensives operates, due to an increase in thickness of only a few millimetres, at an only moderately increased level of wall stress. Because of the cubic relationship between ventricular dimensions and volume, ventri

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Left Ventricular Hypertrophy: Pathology Versus Physiology,se-induced) left ventricular hypertrophy. Physiologic hypertrophy usually occurs in highly-trained athletes and can be considered as a normal adaptation to a chronic pressure or volume overload. Pathologic left ventricular hypertrophy is the result of maladaptation of the heart to overload and is a

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Hypertrophy and Arrhythmias,genic potential is most likely based on several factors, including significant electrophysiological alterations, anatomic alterations (fibrosis) and increased propensity for ischemic events. There is no single responsible arrhythmogenic mechanism. Hence, anti-arrhythmic therapy will be hard to manag

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Left Ventricular Hypertrophy: Reversal by Antihypertensive Therapy,h LVH results in lowering of blood pressure and drug-dependent reduction of LV mass. Complete reversal of LVH can be accomplished in 2–3 years using angiotensin converting enzyme (ACE)-inhibitors. Novel drugs with profound inhibitory effects on increased LV mass are angiotensin-II antagonists. The r