Titlebook: Integration/Interaction of Oncologic Growth; Gary G. Meadows Book 20051st edition Springer Science+Business Media B.V. 2005 Chemokine.angi

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Influence of the Bone Microenvironment on Breast Cancer Metastasis to Bone,roma interactions may identify critical factors regulating metastatic progression and lead to the development of stromal therapies for breast and other malignancies. Here, the evidence implicating stromal factors in the metastasis of breast tumours to bone will be reviewed.

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Interaction of Tumour with Host Stroma in Hepatocellular Carcinoma,e tumor, a kind of host defense mechanism. It is unlikely to be as simple as this, since the presence of the capsule may reflect inherently attenuated invasive properties of the tumor. This chapter reviews the interactions between hepatocellular cancer and hepatic stroma, and reveals a complex model

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Dynamic Nature of Tumour-Host Interactions Within the Tumor Microenvironment,e to the execution of their effector function. In fact, metastatic melanoma stands out among human cancers because of its immune responsiveness. Yet, the reason(s) remain(s) unclear. We believe that the key to the understanding of this complex phenomenon relies on the real-time study of tumor/host i

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,Breast Cancer Progression: A “Vicious Cycle” of Pro-Malignancy Activities is Mediated by Inflammatoter, major emphasis is put on the manner by which the different inflammatory mediators cross-interact with each other, as well as with the tumor cells, together establishing an inflammatory microenvironment that consists of an extensive network of pro-malignancy activities.

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Tumor and Host Endothelial Cell Selective Interactions and Modulation by Microenvironmental Chemokifined cell and molecular interactions are insured by sugar-mediated recognitions since glycosylation is the post-translational transformation that confers to glycoconjugates their structural specific identity as recognitions signals. Advantage could be taken of such specific characters as a way to d

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Integrin-Mediated Resistance to Chemotherapy-Induced Apoptosis in Cancer Cells, chemotherapy. In this chapter we review the work that has been undertaken to elucidate the mechanisms underlying this phenomenon. The identification of signalling pathways underlying integrin-mediated drug resistance offers the possibility of designing novel therapeutic agents that could be adminis

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