Titlebook: Insulin Resistance and Polycystic Ovarian Syndrome; Pathogenesis, Evalua Evanthia Diamanti-Kandarakis,John E. Nestler,Renat Book 2007 Human

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Serine Phosphorylation, Insulin Resistance, and the Regulation of Androgen Synthesis,. It is likely that PCOS is a group of distinct diseases with similar clinical phenotypes but different pathophysiological mechanisms, rather than being one disease caused by a single molecular defect. The . can potentially explain two major features of the syndrome: insulin resistance and hyperandr

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Adrenarche,no longer appears to be accurate. In adults, a major risk factor for development of coronary artery disease and type 2 diabetes is the presence of MS characterized by insulin resistance (IR), glucose intolerance, dyslipidemia, hypertension, and central obesity and, in women, the added feature of PCO

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Late Pregnancy Complications in Polycystic Ovary Syndrome,ncy. Many features of the insulin resistance syndrome have been associated with these conditions. These include glucose intolerance, hyper-insulinemia, hypertension, obesity, and lipid abnormalities. Other accompanying abnormalities may include elevated serum levels of leptin, tumor necrosis factor-

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