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Titlebook: Improvement of Myocardial Perfusion; Thrombolysis, angiop Jürgen Meyer,Raimund Erbel,Hans Jürgen Rupprecht Book 1985 Springer Science+Busin

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H.-J. Rupprecht,R. Erbel,K.-H. Schöter,G. Schreiner,K.-L. Hennrichs,J. Meyernd that most contributions only scratched at the surface of Collaborative Planning. Most frequently, discussions focused on the technological means available for the exchange of information - tween independent Supply Chain partners, such as Email, Web-interfaces, or the XML technology.
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Coronary venous retroperfusion support of reperfusion following acute coronary occlusionlloon occlusion and reperfusion. Supplementation of retroperfusion with streptokinase retroinfusion via the great cardiac vein was found to provide retrograde lysis of an occlusive trombus instituted in the LAD coronary artery of dogs. Retroperfusion and retroinfusion are believed to offer substanti
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Coronary thrombolysis with tissue-type plasminogen activator (t-PA)better when an occluded coronary artery is reopened within at most a couple of hours [4]. The proof that coronary reperfusion is of real benefit to patients with acute myocardial infarction in terms of morbidity or mortality is however still lacking [5–7]. To this end several large-scale trials are currently in progress.
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Opposite ends of the intervention spectrum: Effects of intracoronary thrombolysis on long-term survi part of therapy aimed at regression — a process we call diagnostic therapeutics. We believe that the evolution of these two approaches at the opposite ends of intervention cardiology will radically and profoundly change the field by becoming routine clinical practice over the next five years.
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Book 1985Sep­ tember 27-29,1984 in Mainz/Germany. It has been the purpose of this meeting to focus the interest of scientifically and clinically interested cardiologists on the new developments in this field. We therefore chose the subtitle ‘Medical-Mechanical -Surgical Approach‘. The medical improvements in
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Limitation of myocardial necrosis by thrombolysis during acute myocardial infarctionyocardial necrosis, efforts were focused on the development of interventions that would limit the extent of necrosis and reduce infarction size. Although several of these interventions appeared to be effective in experimental studies, they proved to have little or no impact in clinical practice [2].
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