Titlebook: Immunoneurology; Michel Chofflon,Lawrence Steinman Book 1996 Springer-Verlag Berlin Heidelberg 1996 Guillian Barr syndrom.cytokine.cytokin

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共同确定为确

Superantigens in demyelinating disease,d in vivo or in vitro. This process leads to the clonal expansion of autoreactive cells. In addition, during the initiation phase of demyelinating disease, only activated autoreactive lymphocytes migrate through the endothelial cell layer of the intact blood-brain barrier (BBB).

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Are current immunological concepts of multiple sclerosis reflected by the immunopathology of its le the lesions may be fundamentally different in individual MS patients and that autoimmunity may not be the sole cause. In the case of autoimmune reactions various different proteins of the nervous system may become targets and it appears unlikely, that myelin basic protein is a major candidate for a pathogenetic role in MS.

embolus

Autoimmune responses in peripheral nerve,neuropathies and possibly paraneoplastic neuropathies. Here we attempt to present an overview of recent advances in understanding immune responses in the peripheral nervous system (PNS) that underlie these disorders.

仇恨

Immune control of the brain, CNS cells as antigen-presenting cells. Upon contact with “their” specific antigens, they initiate local immune responses. Then, in a second step, cells of the local glia and of the immune system communicate bidirectionally with each other and interact to jointly control the immune reactivity within the CNS.

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