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派博传思国际中心 »期刊书目

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Titlebook: Immunology and Immunopathogenesis of Malaria; Jean Langhorne Book 2005 The Editor(s) (if applicable) and The Author(s), under exclusive li

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发表于 2025-3-21 16:06:00 | 显示全部楼层 |阅读模式
书目名称Immunology and Immunopathogenesis of Malaria
编辑Jean Langhorne
视频videohttp://file.papertrans.cn/463/462215/462215.mp4
概述Addresses vital questions of immunopathology surrounding malaria, which kills more than 1 million people annually.Discusses drug resistance of the parasite, insecticide resistance of the mosquito, and
丛书名称Current Topics in Microbiology and Immunology
图书封面Titlebook: Immunology and Immunopathogenesis of Malaria; Jean Langhorne Book 2005 The Editor(s) (if applicable) and The Author(s), under exclusive li
描述.Malaria is still a major global health problem, killing more than 1 million people every year. Almost all of these deaths are caused by Plasmodium falciparum, one of the four species of malaria parasites infecting humans. This high burden of mortality falls heavily on Sub-Saharan Africa, where over 90% of these deaths are thought to occur, and 5% of children die before the age of 5 years. The death toll from malaria is still growing, with malaria-specific mortality in young African children estimated to have doubled during the last twenty years. This increase has been associated with drug resistance of the parasite, spread of insecticide resistant mosquitoes, poverty, social and political upheaval, and lack of effective vaccines...This collection of reviews addresses many of these important issues of malarial immunity and immunopathology. They are of interest not only to malariologists, but hopefully also to the broader immunological community. Strong interactions with, and feedback from immunologists working in other infectious diseases and in basic immunology will help us to move the field of malaria immunology and therapeutic intervention forward more quickly..
出版日期Book 2005
关键词Malaria; anemia; blood; blood stage malaria infection; diseases; drug resistance; infection; infectious; inf
版次1
doihttps://doi.org/10.1007/3-540-29967-X
isbn_softcover978-3-642-42184-6
isbn_ebook978-3-540-29967-7Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
The information of publication is updating

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Current Topics in Microbiology and Immunologyhttp://image.papertrans.cn/i/image/462215.jpg
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978-3-642-42184-6The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
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Immunology and Immunopathogenesis of Malaria978-3-540-29967-7Series ISSN 0070-217X Series E-ISSN 2196-9965
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The Dissection of CD8 T Cells During Liver-Stage Infection,. Malaria antigen (Ag)-specific CD8 T cells that produce IFN-γ are key effector cells in this model of protection. Although there have been numerous reports dealing with γ-spz-induced CD8 T cells in the spleen, CD8 T cells most likely confer protection by targeting infected hepatocytes. Consequently
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Early Interactions Between Blood-Stage , Parasites and the Immune System,. parasites, the causative agents of malaria. Early interactions between blood-stage parasites and cells of the innate immune system, including dendritic cells, monocytes/macrophages, natural killer (NK) cells, NKT cells, and γδ T cells, are important in the timely control of parasite replication an
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Longevity of the Immune Response and Memory to Blood-Stage Malaria Infection, of protective immune responses are sparse.However, studies of antibody responses associated with protection reveal that they consist of a short- and a long-lived component. Compared with the antibody levels observed in other infection and immunization systems, the levels of the short-lived antibody
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Glycosylphosphatidylinositols in Malaria Pathogenesis and Immunity: Potential for Therapeutic Inhibf ., but may also exist free of protein attachment. In vitro and in vivo studies have established GPIs as likely candidate toxins in malaria, consistent with the prevailing paradigmthat attributes induction of inflammatory cytokines, fever and other pathology to parasite toxins released when schizon
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