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Titlebook: Immune Mechanisms in Renal Disease; Nancy B. Cummings,Alfred F. Michael,Curtis B. Wils Book 1983 Springer-Verlag US 1983 DNA.allergy.antig

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书目名称Immune Mechanisms in Renal Disease
编辑Nancy B. Cummings,Alfred F. Michael,Curtis B. Wils
视频video
图书封面Titlebook: Immune Mechanisms in Renal Disease;  Nancy B. Cummings,Alfred F. Michael,Curtis B. Wils Book 1983 Springer-Verlag US 1983 DNA.allergy.antig
出版日期Book 1983
关键词DNA; allergy; antigen; blood; cancer; diseases; fluorescence; glomerulonephritis; immunology; inflammation; ki
版次1
doihttps://doi.org/10.1007/978-1-4613-4625-8
isbn_softcover978-1-4613-4627-2
isbn_ebook978-1-4613-4625-8
copyrightSpringer-Verlag US 1983
The information of publication is updating

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Pathologic and Functional Correlations in the Glomerulopathies,d also could provide a useful tool with which to assess the response of patients to treatment. Although the measurement of protein selectivity (Joachim .., 1964; Cameron and Blandford, 1966) has gained some popularity for such purposes, there are many inherent problems in this method (Pesce .., 1970). Thus, it has not gained widespread acceptance.
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Deposition and Removal of Glomerular Immune Complexes: Relationships to the Mononuclear Phagocyte S well documented also. The estimate has been made that over 90% of acute glomerulonephritis is caused by immune complexes. The reasons for the variations in the course and prognosis of glomerulonephritis are not known adequately at this time.
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Autoimmune Disease Induced in Rabbits by Administration of Mercuric Chloride: Evidence Suggesting a in man. Data available suggest the involvement of antigens of glomerular basement membrane (GBM), cell nuclei, thyroid, and, possibly, brush border of proximal tubules, and tumors (Wilson and Dixon, 1976). The mechanisms of autoimmunization, however, are not well understood.
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Nancy B. Cummings,Alfred F. Michael,Curtis B. Wils
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