Titlebook: Hypertension: Mechanisms and Management; Thomas Philipp,Armin Distler Conference proceedings 1980 Springer-Verlag Berlin Heidelberg 1980 A

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G. Schmid,A. Heidlandlation, reperceiving, and the Intention, Attention, Attitude (IAA) triaxiomatic model. Subsequent chapters discuss mindfulness-based interventions and mechanisms of change for stress and related clinical con978-3-030-81247-8978-3-030-81245-4

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F. H. M. Derkx,B. N. Bouma,H. L. Tan-Tjiong,A. J. Man in’t Veld,J. H. B. de Bruyn,G. J. Wenting,M. Aapproaches.Arab American health disparities: A call for advocacy. .Rich in cultural information and clinical insights, .Biopsychosocial Perspectives on Arab Americans. is an important reference that can enhance978-1-4899-7639-0978-1-4614-8238-3

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Cardiovascular Pressor Reactivity as Related to Plasma Catecholamines: Role in the Pathogenesis of E 0.001) in established hypertension; it correlated inversely (P < 0.01) with basal blood pressure..Adrenergic neuronal blockage with debrisoquine decreased plasma NE by 50% in all three groups; blood pressure remained largely unchanged in normal subjects, decreased only slightly (-5%) in borderline

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Blunting of Beta-Adrenoceptor-Mediated Cardiovascular Responses and Increasing Alpha-Receptor-Mediatreceptor-related effect tends to be enhanced as judged from the greater reduction of cardiac isoproterenol sensitivity and the blunted renin response to exercise stimulation..The dilator response to alpha-adrenoceptor blockade with phentolamine was not different in both groups. Therefore a qualitati

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Round Table Discussion: Does the Renin-Angiotensin System Play a Signigicant Role in Human Hypertensge of those who spoke this morning on catecholamines and the sympathetic nervous system. This demonstrates that most of us here who present data this afternoon have been in the field already for a long time. So we are an “old generation” of hypertensiologists whereas this morning a “younger generati

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Urinary Kallikrein Activity in Rats with Renovascular Hypertensionis probably not involved in the development of renovascular hypertension and [2] that in severe renal hypertension renal kallikrein activity is not only reduced in urine of the ischaemic kidney, but also in urine of the contralateral one.

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0173-0282 Overview: 978-3-540-10171-0978-3-642-67712-0Series ISSN 0173-0282