Titlebook: Hormones in Lipoprotein Metabolism; Armin Steinmetz,Jürgen Schneider,Hans Kaffarnik Conference proceedings 1993 Springer-Verlag Berlin Hei

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Lipoproteins in Patients with NIDDM: Effects of Different Therapeutic Approachess with NIDDM. According to the work of Reaven and his group, an increase in VLDL secretion occurs with decreasing insulin response, the second fact is that actual serum levels of triglycerides (TG) are higly determined by VLDL secretion. The increase in VLDL production seems to depend mainly from a

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Postprandial Course of Triglycerides and the Atherogenic Lipoprotein Particle “IDL” in Type 2 Diabetributing to the increased prevalence of atherosclerosis in these patients. In the Paris Prospective Study, hypertriglyceridemia was found to be an independent risk factor for coronary heart disease in normocholesterolemic diabetic patients [3]. More recently, these epidemiological data were confirme

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Differences between Basal and Postprandial Circadian Variation of Insulin Sensitivity in Healthy Subents treated with a glucose-controlled insulin infusion system. In both groups the postprandial glycemic response to identical low caloric mixed meals ingested at 8 a. m., 1 p. m., and 6 p. m. increased significantly in the course of the day. In contrast to findings in conventional insulin therapy m

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Effect of Fish Oil Concentrate on the Lipoprotein Profile in Patients Suffering from Diabetes mellit the increased atherogenic risk in these patients [1, 6]. The endogenous synthesis of the very-low-density (VLDL)-triglycerides [13] and of the apolipoprotein B (apo B) [4] is increased and results in a pathological serum level of triglycerides. Low-density-lipoprotein (JLDL)-apo B is also raised an

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Adrenal-Glands

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Changes in Serum Lipoprotein Profiles During Puberty — A Longitudinal Study of Dresden Schoolchildre lower HDL-C, Apo Al, HDL-2C and HDL-3C in males, whereas no statistically proved differences in regard to serum concentrations of Apo All, D, E could be observed in our and other studies [1, 5], It is commonly supposed that these differences are the result of sexual maturation. With respect to the

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Coronary Risk in Women: Potential Role of Age, Lipoproteins, Haemostasis, and Hormonesinfarction (Ml) in the fifth and sixth decades of life, whereas during this period incidence of Ml is low in women but increases dramatically after age 55. The reason for these sex-specific differences in the development of coronary heart disease (CHD) is currently unknown but has mostly been adress