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Titlebook: Hormonal Carcinogenesis III; Proceedings of the T Jonathan J. Li (Professor of Pharmacology, Toxicol Conference proceedings 2001 Springer-V

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楼主: 遮阳伞
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The Effects of Triphenylethylene Antiestrogens on Parameters of Multistage Hepatocarcinogenesis in tenetic and/or epigenetic manner. In animals, the stages of initiation, promotion, and progression can be operationally demonstrated (1,2). The rat liver has been extensively analyzed as a multistage model system for analysis of the stage(s) at which a compound can act to modify the carcinogenic proc
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Introductionrogen treatment has essentially replaced endocrine ablation as first line therapy for advanced breast cancer (BC) of the hormone-dependent kind (2). These so-called type I agents show curious pharmacological behavior. Depending on species, tissue, and concentration, these compounds, and their active
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Estrogen Receptor-ß: An Important Player in Estrogen Actionne single estrogen receptor (ER) mediating all effects of estrogenic hormones led to severe limitations in our understanding of estrogen action, for instance with reference to tissue specific hormonal effects. The entrance of a new ER with slightly different ligand binding specificity than the “old”
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Estrogen Receptor-mediated Genomic Instability in the Syrian Hamster Kidney: A Critical Event in Horn receptor (ER-α)-mediated cell proliferation. The focus of the studies presented herein is to provide evidence for this contention, and for a multi-stage sequence of events leading to tumor development, employing the Syrian hamster E-induced and -dependent renal neoplasm model.
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Jonathan J. Li (Professor of Pharmacology, Toxicol
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Yvonne P. Dragan,Emile Nuwaysir,Linda Sargent,Dong-Hui Li,V. Craig Jordan,Henry C. Pitot
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Jonathan J. Li,S. John Weroha,Marilyn Cansler,Sara Antonia Li
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