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Titlebook: Highlights in Asthmology; François Bernard Michel,Jean Bousquet,Philippe God Book 1987 Springer-Verlag, Berlin Heidelberg 1987 allergy.ant

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楼主: Malicious
发表于 2025-3-26 21:36:49 | 显示全部楼层
M. Perrin-Fayolleological, epidemiologic, preventi­ ve and therapeutic aspects. With the cooperation of an outstanding group of inter­ national lecturers we have been able to present an overview of the disease and its accidents as it stands today. Such a scope differs greatly from the narrow workshops which only con
发表于 2025-3-27 04:56:05 | 显示全部楼层
ological, epidemiologic, preventi­ ve and therapeutic aspects. With the cooperation of an outstanding group of inter­ national lecturers we have been able to present an overview of the disease and its accidents as it stands today. Such a scope differs greatly from the narrow workshops which only con
发表于 2025-3-27 08:18:20 | 显示全部楼层
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C. W. Parker, restenosis remains an ever-present limitation of all angioplasty modes including stent application (1,17–19,29,36,39,42,45,46). This problem is mainly due to our still incomplete concepts of angioplasty-induced healing and growth processes in the vascular wall. As a result, effective causal therap
发表于 2025-3-27 17:30:16 | 显示全部楼层
A. L. de Weckdworldwide per year. However, the enthusiasm forangioplasty is shadowedby restenosis, which remains a criticalproblem after the procedure,resulting in clinical recurrence in overa third of patients. Recently, the mechanisms of restenosis have beencompletely reappraised with anenriched understanding
发表于 2025-3-27 18:11:34 | 显示全部楼层
W. E. Parishhe failure to inhibit restenosis raises questions about the pathophysiology classically used to identify restenosis, i.e., smooth muscle cell proliferation (1–4). It is well established that balloon angioplasty commonly induces a neointimal formation with smooth muscle migration from the media, and
发表于 2025-3-28 00:17:38 | 显示全部楼层
M. Ricci,E. Maggi,G. F. del Prete,S. Romagnanirestenosis after percutaneous transluminal coronary angioplasty (PTCA) and hypertension. Glagov (1) was one of the first to demonstrate that the atherosclerotic vessel attempted to “accommodate” increasing plaque burden by enlarging vessel diameter based on studies of patient necropsy samples. Recen
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