Titlebook: High Altitude; Human Adaptation to Erik R. Swenson,Peter Bärtsch Book 2014 Springer Science+Business Media New York 2014 Acute Mountain Si

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Control of Breathing,rotid bodies containing oxygen sensors), followed immediately by a secondary roll-off that partly is caused by the hyperventilation-induced hypocapnia. When the hypoxia is sustained, ventilation starts to rise gradually within the next few hours, that, depending on the species and altitude (degree o

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Lung Function and Gas Exchange, function at high altitude. Environmental hypoxia, combined with cold and heavy exercise all contribute to considerable stress to the lung. Acute altitude exposure results in hypoxia, and a rapid and sustained increase in alveolar ventilation, with an associated fall in alveolar and arterial partial

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Skeletal Muscle Tissue Changes with Hypoxia,e (>5,500 m). There is consensual evidence that continued sojourn at these altitudes has a number of negative consequences to muscle tissue. There is a loss of muscle mass related to a decrease of individual muscle fiber cross-sectional area. There is also a relative and absolute decrease in muscle

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Blood and Haemostasis,cell mass, particularly in the various settings used by athletes. Furthermore, mechanisms of neocytolysis occurring on descent from high altitude are discussed, as well as mechanisms accounting for a rapid decline of erythropoietin serum levels during persistent hypoxia, whilst increased erythropoie

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Renal Function and Fluid Homeostasis,modynamics all play a far greater part in altering renal function and fluid balance. Acute and chronic effects of hypoxia and their magnitude may differ and these will be highlighted. These changes will be related to the common diseases of high altitude and to their impact on patients with chronic r