Titlebook: Handbuch Frieden; Hans J. Gießmann (Direktor),Bernhard Rinke (Wissen Book 20111st edition VS Verlag für Sozialwissenschaften | Springer Fa

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nism-based preclinical pharmacological intervention used in animal models. Their effects on cell death, inflammatory events, and prolonged motor and cognitive deficits will be summarized, and their potential success for clinical application will be evaluated. Many of the above-mentioned mechanisms m

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Hans J. Gießmann,Bernhard Rinkee metabolic profile of the affected tissue is significantly altered, with the infarct representing the most severely affected tissue, and the surrounding penumbra, or peri-infarct zone (PIZ), containing a gradient of metabolic states progressing from severely impacted toward an otherwise healthy pro

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Reinhard Meyersspinal cord trauma, hypoglycemic coma, and prolonged epileptic seizures. All of these lead to high concentrations of calcium in nerve cells which, in turn, causes degradation of cytoplasmic proteins, cleavage of nuclear DNA, and eventually cell death. .The Second Edition contains 11 thoroughly updat

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am effector mixed lineage kinase domain-like (MLKL). Necroptotic signals induced by death receptors, such as TNF receptor 1, Toll-like receptors or interferon receptors lead to the formation of the necrosome, and result in cell death with morphological features of necrosis. The execution of necropto

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Sabine Jabergemingly disparate, stroke, brain and spinal cord trauma, coma from a low serum glucose concentration (hypoglycemia), and prolonged epileptic seizures have in common the inciting factor of excitotoxicity, the activation of a specific subtype of glutamate receptor by an elevated extracellular glutamat

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Sabine Jabergemingly disparate, stroke, brain and spinal cord trauma, coma from a low serum glucose concentration (hypoglycemia), and prolonged epileptic seizures have in common the inciting factor of excitotoxicity, the activation of a specific subtype of glutamate receptor by an elevated extracellular glutamat

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Jean-Daniel Strubemingly disparate, stroke, brain and spinal cord trauma, coma from a low serum glucose concentration (hypoglycemia), and prolonged epileptic seizures have in common the inciting factor of excitotoxicity, the activation of a specific subtype of glutamate receptor by an elevated extracellular glutamat

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