Titlebook: Handbook of Neurochemistry and Molecular Neurobiology; Degenerative Disease Abel Lajtha (Director),Moussa B. H. Youdim (Profes Reference wo

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,Huntington’s Disease: Unraveling the Pathophysiological Cascade Behind a Singular Gene Defect,defect, i.e., a CAG triplet expansion that is translated into polyglutamine chains within different proteins. Although HD is caused by a singular gene defect, this disease shows a high variability in age of onset and in its clinical manifestations, which include characteristic motor, cognitive, and

homeostasis

,Dementia: The Significance of Cerebral Metabolic Disturbances in Alzheimer’s Disease. Relation to Pical entities. A minority of all AD cases is caused by mutations on chromosomes 1, 14 and 21, a minority of all PD cases by mutations on chromosomes 1, 2, 4, 6 and 12, whereas the majority of all both AD and PD cases is age‐related and sporadic in origin. In sporadic AD, the desensitization of the n

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,Proteomics Analysis in Alzheimer’s Disease: New Insights into Mechanisms of Neurodegeneration,ometry and bioinformatics. Using redox proteomics, brain and plasma proteins in Alzheimer‘s disease (AD) and models thereof that are oxidatively modified have been identified. Most oxidatively modified proteins are dysfunctional, suggesting that in AD the function of oxidatively modified proteins is

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