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Titlebook: Handbook of Cancer Vaccines; Michael A. Morse,Timothy M. Clay,H. Kim Lyerly Book 2004 Springer Science+Business Media New York 2004 Antige

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书目名称Handbook of Cancer Vaccines
编辑Michael A. Morse,Timothy M. Clay,H. Kim Lyerly
视频video
概述Includes supplementary material:
丛书名称Cancer Drug Discovery and Development
图书封面Titlebook: Handbook of Cancer Vaccines;  Michael A. Morse,Timothy M. Clay,H. Kim Lyerly Book 2004 Springer Science+Business Media New York 2004 Antige
描述An authoritative survey of the scientific background for therapeutic cancer vaccines, the challenges to their development, and their current uses in treating cancer. The authors examine the basic issues that effect all vaccines (such as immune adjuvants and prime-boost strategies), describe the methods for antigen discovery, and review the preclinical development phases for each major vaccine strategy. They also spell out the clinical results for cancer vaccines now beginning to be used in the treatment of many common cancers.
出版日期Book 2004
关键词Antigen; DNA; cancer; cell; clinical trial; cytokine; cytokines; immunotherapy; melanoma; ovarian cancer; pros
版次1
doihttps://doi.org/10.1007/978-1-59259-680-5
isbn_softcover978-1-4684-9818-9
isbn_ebook978-1-59259-680-5Series ISSN 2196-9906 Series E-ISSN 2196-9914
issn_series 2196-9906
copyrightSpringer Science+Business Media New York 2004
The information of publication is updating

书目名称Handbook of Cancer Vaccines影响因子(影响力)




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书目名称Handbook of Cancer Vaccines网络公开度




书目名称Handbook of Cancer Vaccines网络公开度学科排名




书目名称Handbook of Cancer Vaccines被引频次




书目名称Handbook of Cancer Vaccines被引频次学科排名




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书目名称Handbook of Cancer Vaccines年度引用学科排名




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书目名称Handbook of Cancer Vaccines读者反馈学科排名




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urnet and Lewis Thomas . has been recently highlighted in experiments with gene knockout mice. Compared with wild-type mice, mice lacking sensitivity to either interferon-γ (IFN-γ receptor-deficient mice) or all interferon (IFN) family members (i.e., Stat 1 -deficient mice) developed tumors more rap
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https://doi.org/10.1057/9780230119819nes. Improved understanding of the molecular basis of antigen recognition has resulted in the development of rationally designed tumor antigen—specific vaccines based on motifs predicted to bind to human class I or class II major histocompatibility complex (MHC) molecules. The use of synthetic pepti
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