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书目名称Growth Hormone and Somatomedins during Lifespan影响因子(影响力)




书目名称Growth Hormone and Somatomedins during Lifespan影响因子(影响力)学科排名




书目名称Growth Hormone and Somatomedins during Lifespan网络公开度




书目名称Growth Hormone and Somatomedins during Lifespan网络公开度学科排名




书目名称Growth Hormone and Somatomedins during Lifespan被引频次




书目名称Growth Hormone and Somatomedins during Lifespan被引频次学科排名




书目名称Growth Hormone and Somatomedins during Lifespan年度引用




书目名称Growth Hormone and Somatomedins during Lifespan年度引用学科排名




书目名称Growth Hormone and Somatomedins during Lifespan读者反馈




书目名称Growth Hormone and Somatomedins during Lifespan读者反馈学科排名

渐变

BRAWL

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CHIDE

,Vorbedingungen für die Planung, the authors’ laboratory using several transgenic and genetic strains of mice and rats with altered function of the individual axis components. These functional changes all produce profound effects on growth as well as on other components of the hypothalamic-pituitary axis.

monologue

Studies on Peptide 23: A GHRH-Regulated Protein of the Pituitary and Gastrointestinal Tractbe paracrine regulators of GH secretion. However, as pointed out by O’Halloran et al. (1991), a number of conditions must be met to establish a paracrine role for any of the putative regulatory factors. As well the influence may not only be on secretion of GH but also on GH cell proliferation or differentiation.

monologue

Insulin-like Growth Factor Binding Proteins: Biochemical Characterization these proteins, both in their primary structure and resulting from post-translational modification, which contribute to their unique biological functions. Of necessity, the review cannot be comprehensive, but will be illustrated by selected examples, including studies conducted in the author’s laboratory.

Hypomania

Autocrine and Paracrine Effects of Insulin-like Growth Factors and Their Binding Proteinss are devoid of IGF-I receptors (Caro et al. 1988). If the liver is the major producer of IGF-I, the lack of IGF-I mitogenic effect on hepatocytes is a support for the endocrine hypothesis of IGF-I mode of action.

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https://doi.org/10.1007/978-981-13-2242-6ters whose dual effect on GH secretion can be explained by the action of the same molecule either at the level of GHRF- or SRIF-secreting neurons (Fig. 1). This is well exemplified by catecholamines, where α. and α.-β. adrenoceptors exert respectively stimulatory and inhibitory influences on GH secretion (Müller et al. 1991).