Titlebook: Glial ⇔ Neuronal Signaling; Glenn I. Hatton,Vladimir Parpura Book 2004 Springer Science+Business Media New York 2004 biology.brain.cell.ce

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Astrocyte gap junctions and glutamate-induced neurotoxicity,ynapses (Bennett, 1977) and provide the intercellular pathway for astrocytic networks (Giaume and McCarthy, 1996). We have recently reviewed the expression of gap junctions in the central nervous system and discussed their physiopathological relevance in several brain dysfunctions (Rouach et al., 20

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Glutamate-mediated bi-directional signaling between neurons and astrocytes, neurons and astrocytes intermingle forming a morphologically intimate relationship (Chapters 3 and 4) where astrocytes are favorably positioned to exchange signals with neurons (Figure 15.1). Ca. entry though voltage-gated channels into the presynaptic terminal signals to the secretory machinery, w

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Book 2004 interactions. Even experts in the field will find useful the authoritative summaries of evidence on ion channels and transporters in glia, genes involved in signaling during development, metabolic cross talk and cooperation between astrocytes and neurons, to mention but a few of the timely summarie

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https://doi.org/10.1007/978-3-642-25702-5lated astrocytes release glutamate, which by both pre- and post- synaptic mechanisms modulates synaptic strength. The ability of astrocytes to sense neuronal activity and respond by increases in [Ca.], has been a topic of intense research for the last few years. Here we review the mechanisms of astrocytic Ca. signaling.

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