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Titlebook: Glaucoma Update II; Glaucoma Society of G. K. Krieglstein,W. Leydhecker Conference proceedings 1983 Springer-Verlag Berlin Heidelberg 1983

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,Disc Haemorrhages — Forerunners of Chronic Glaucoma, years the results have appeared from survey material and glaucoma follow up studies (Bengtsson et al. 1980, 1981; Holmin and Krakau 1981) and of retrospective investigations (Airaksinen et al. 1981, Gloster 1981).
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The Mechanisms of Damage of the Optic Nerve,e possible (1) to devise clinical tests to predict those individuals who are at risk of developing nerve damage and (2) to devise therapeutic measures that can be directed at factors other than intraocular pressure that pertain to the pathophysiologic process by which the optic nerve is damaged in glaucoma.
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Kai Kratzer,Dominic Roehm,Axel Arnoldiveness of these drugs for the treatment of glaucoma is now well established. Not only the knowledge of the mechanism of their action, but also of pharmacokinetics is of value for designing logical therapeutic regimen. Therefore, the recent findings on the latter aspects of the ß-adrenergic drugs ar
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Wolfgang E. Nagel,Willi Jäger,Michael Reschndicated abnormalities in the drainage routes. It would be natural then to start therapy by attempts at increasing the facility and the ideal drug, of course, would be one that restituted facility to its normal level and the outflow routes to their normal appearance. The search for such an agent is
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Martin Karrenbach,Petra Knopf,Matthias Jacobts were made by rapidly (0.2 s) injecting indocyanine green dye (2.5 mg/ml), in 0.1 ml increments, into the common carotid artery and photoelectrically monitoring its concentration in the superior anterior ciliary vein. In the six normal cats studied, the choroidal blood flow during the control peri
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