Titlebook: Genomic Instability and Immortality in Cancer; Enrico Mihich,Leland Hartwell Book 1997 Springer Science+Business Media New York 1997 DNA.M

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书目名称Genomic Instability and Immortality in Cancer影响因子(影响力)




书目名称Genomic Instability and Immortality in Cancer影响因子(影响力)学科排名




书目名称Genomic Instability and Immortality in Cancer网络公开度




书目名称Genomic Instability and Immortality in Cancer网络公开度学科排名




书目名称Genomic Instability and Immortality in Cancer被引频次




书目名称Genomic Instability and Immortality in Cancer被引频次学科排名




书目名称Genomic Instability and Immortality in Cancer年度引用




书目名称Genomic Instability and Immortality in Cancer年度引用学科排名




书目名称Genomic Instability and Immortality in Cancer读者反馈




书目名称Genomic Instability and Immortality in Cancer读者反馈学科排名

STALE

sleep-spindles

Aneuploidy and Heterogeneity Mechanisms in Human Colorectal Tumor Progression,ics and, more recently, by chromosome painting and interphase cytogenetics using fluorescence in situ hybridization (12,13), appears simply too difficult to be set in a cause-effect relationship. An initial preneoplastic genetic event predisposing to more generalized disruption of the genome has bee

CLEAR

p53-Dependent Signaling in Response to DNA Damage or Arrest of DNA Synthesis and its Role in Cell Cls arrest late in G1 when p53 is active. In addition to G1 arrest, regulation by p53 of the other major cell cycle checkpoint, G2/M, has recently been reported in two different systems (Stewart ., 1995; Agarwal .., 1995).

NIL

Cardioplegia

Cardioplegia

Regulation of Telomere Length in Mammalian Cells,ons would lead to a progressive loss of DNA. Mammalian telomerase activity was first identified in HeLa cell extracts which can add over sixty T.AG. repeats onto a T.AG. oligonucleotide . [5]. Mouse cell extracts were shown to add at most one or two T.AG. repeats onto a T.AG. oligonucleotide . sugge

TERRA

名义上

,Neoplastic Progression in Barrett’s Esophagus,d carcinomas in surgical specimens. Although the study of advanced cancers is useful for identifying genetic abnormalities that have accumulated in the cancer, the ability of this approach to determine the order in which genetic and other abnormalities develop during earlier stages of neoplastic pro

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