Titlebook: Genome Stability and Human Diseases; Heinz-Peter Nasheuer Book 2010 Springer Science+Business Media B.V. 2010 Chromosom.DNA.Viruses.genes.

CLAM

,Einige Regeln für Anfragen auf Krane,rring processes such as DNA replication and recombination can result in DNA double-strand breaks (DSBs). DSBs are potentially lethal and so eukaryotic cells have evolved an elaborate pathway, the DNA damage response, which detects the damage, recruits proteins to the DSBs, activates checkpoints to s

FLAIL

Deutsches Krebsforschungszentrum Heidelberg are frequently post-translational modified. Such covalent additions to histone residues can modulate chromatin folding and/or provide specificity to docking surfaces for non-histone chromatin proteins. In the budding yeast, one such modification, transient acetylation of histone H3 on residue lysin

BARK

Wirtschaftliches; Speise und Trankg of DNA damage events and by acting as a foundation for the assembly of repair foci. The H2AX protein sequence is highly similar and in some cases overlapping with replication-dependent canonical H2A, yet the H2AX gene and protein structures exhibit a number of features specific to the role of this

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https://doi.org/10.1007/978-3-662-69378-0and the origin of these mutations and how replication proteins modulate this process. DNA polymerase γ (pol γ) is the polymerase that is responsible for replication and repair of mtDNA. Pol γ has three main roles in mtDNA maintenance and mutagenesis. As the only known DNA polymerase in mitochondria,