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https://doi.org/10.1007/978-3-663-19800-0in the circulation of the host, is apparently a function of the source of the preparation [5]. In early attempts to detect the difference between two such samples (one from Bayer and one from Squibb), each was subjected to gel filtration on a G-200 Sephadex column. Identical activity patterns were o

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https://doi.org/10.1007/978-3-662-64730-1cells has been the subject of numerous presentations at this symposium. The purpose of this report is to describe an automated method for the assay of L-asparaginase and to present data concerning the distribution and clearance of administered L-asparaginase.

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https://doi.org/10.1007/978-3-322-83373-0nt for its characterization, but that as much as possible should be known about its fate in the body — i. e. absorption, blood levels, distribution, elimination, and biotransformation. In the case of Crasnitin . this is of special importance because therapy with enzymes is a rather new field of mode

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https://doi.org/10.1007/978-3-322-99689-3ce of a non-cytopathic, and otherwise benign replicating agent as an adjunct in asparaginase therapy was somewhat unexpected and to our knowledge has no theoretical antecedent. This virus is variously known as the lactate dehydrogenase-elevating virus, the LDH-virus, the enzymes-elevating virus and

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https://doi.org/10.1007/978-3-642-48089-8and later as preparations of varying degrees of purity from ., showed it to be a unique agent with a very high chemotherapeutic index which is capable of curing some mouse leukemias and tumors at high doses without assistance from host immune mechanisms. Since the drug also has a similar chemotherap

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Lebensstiluntersuchungen der Praxis,useful to study experimentally problems which arose from the clinical use of the enzyme. Consequently we investigated the influence of the following factors on tumor inhibition: dosage, duration of treatment, treatment schedules, treatment of Crasnitin resistant tumors, combined treatment with Crasn

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