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Titlebook: Estrogens and Brain Function; Neural Analysis of a Donald W. Pfaff Book 1980 Springer-Verlag New York Inc. 1980 Brain.Function.Gehirn.Hormo

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https://doi.org/10.1057/9780230601208f nerve cell activity. However, even for small bits of neural tissue and restricted forms of behavior, the numbers of nerve cells to be considered are so large and their connections so complex that all sorts of explanatory hypotheses can be imagined. As a result, in the history of neural and behavio
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https://doi.org/10.1057/9781137463647emale rats were studied before and after complete transection of the spinal cord at low thoracic levels. All sham-operated rats showed strong lordosis reflexes before and after surgery. None of the rats receiving complete spinal transections performed lordosis after surgery, even in tests done 3 mon
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Deirdre Torrance,Pamela S. Angelley estrogen in female rats, estrogenic influences on hypothalamic cells are considered primary. In fact, in a wide variety of vertebrate species, estrogens restore sexual receptivity to castrated females (Kelley and Pfaff, 1978). The accumulation of radioactive estradiol by hypothalamic nerve cells i
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https://doi.org/10.1057/9781403981394with hypothalamic cell groups can disrupt food intake, drinking, temperature regulation, aggressive behavior, emotional responses, maternal functions, sleep, arousal, secretion of hormones from the pituitary, and reproductive behavior. Regarding lordosis, hypothalamic axons descending to the midbrai
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brainstem is obligatory, and its net effect must be to facilitate the behavior. Such descending fibers must run in the anterolateral columns (broadly defined), since these are necessary and sufficient for lordosis (Kow et al., 1977). The critical fibers must reach lumbar levels, since the crucial h
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