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Titlebook: Erythrocytes as Drug Carriers in Medicine; Ulrich Sprandel,James L. Way Book 1997 Springer Science+Business Media New York 1997 blood.clin

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Lincoln Bizzozero Revelez,Andrés Raggiots in increased production of reactive oxygen metabolites and nitric oxide, increased phagocytic uptake, and release of many cytokines including IL-1, IL-6, TNF-α and growth factors (M-CSF, G-CSF). Upon activation macrophages also release lipid mediators (prostaglandins and leukotrienes) and numerous enzymes (1).
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https://doi.org/10.1007/978-3-658-17651-8(EC 2.7.1.20) is lower than that of ADA. Adenosine in excess of physiological levels is degraded by ADA. The major source of 2′-deoxyadenosine is DNA turnover and the main route of its metabolism is deamination by ADA.
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Der japanische Zugriff: Trauma #5nd a slow conversion to 5′-monophosphate (AZT-MP) and to AZT. This azidothymidine homodinucleotide seems to have chemical and biochemical properties enabling its profitable utilization in the erythrocyte-encapsulated form.
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bility (as given by the oxygen equilibrium curves) suggest the electroporation/resealing process as a convenient alternative to the hypotonic-dialysis method for the preparation of ADH- and ALDH-erythrocytes.
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The Entrapment of Polyethylene Glycol-Conjugated Adenosine Deaminase (Pegademase) and Native Adenos(EC 2.7.1.20) is lower than that of ADA. Adenosine in excess of physiological levels is degraded by ADA. The major source of 2′-deoxyadenosine is DNA turnover and the main route of its metabolism is deamination by ADA.
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