Titlebook: Epidemiological and Molecular Aspects on Cholera; T. Ramamurthy,S.K. Bhattacharya Book 2011 Springer Science+Business Media, LLC 2011 infe

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Toxins of , and Their Role in Inflammation, Pathogenesis, and Immunomodulation,lence factors in . has invoked interest in their potential role related to pathogenesis, inflammation, and immune modulation in its disease attributes. Cholera was earlier thought to be a non-inflammatory diarrhea, but several reports provide evidence for an inflammatory response in cholera disease.

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, Hemolysin: An Enigmatic Pore-Forming Toxin, a native monomeric molecular weight of 65,000 and induces colloid osmotic lysis in a wide spectrum of eukaryotic cells including rabbit erythrocytes and human enterocytes by forming transmembrane, β-barrel heptameric pores with an internal diameter of ~1.5 nm. The purified toxin is enterotoxigenic

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Integron-Mediated Antimicrobial Resistance in ,,tment of cholera patients, administration of antimicrobials is a common existing practice as it shortens the volume and duration of diarrhea, duration of hospitalization, and excretion of the causative pathogen. Due to excessive use of antibiotics, many groups of infectious bacteria have become resi

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Management of Cholera, This disease is transmitted mostly by faecal-oral route through contaminated water. Clinically, cholera is characterized by uncontrolled purging of watery stool leading to life-threatening dehydration, hypovolaemic shock, acidosis, and if left untreated, it can also lead to death. Asymptomatic carr

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Al-Si Alloys Casts by Die Castingronmental factors including El Niño were attributed to the increased incidence of cholera. During and after the cholera outbreaks in South America, intensive measures were taken that include epidemiological surveillance, setting up of a laboratory network, proper patient care, improvements in basic

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https://doi.org/10.1007/978-3-031-15004-3oxigenic ., which replaced existing clones, possibly through a natural selection involving unidentified genetic and environmental factors as well as immunity of the host population. The continual emergence of new genetic variants and their selective enrichment during cholera outbreaks appear to cons