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Titlebook: Endocrinology of Cardiovascular Function; Ellis R. Levin,Jerry L. Nadler Book 1998 Springer Science+Business Media New York 1998 Angiotens

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发表于 2025-3-21 18:52:52 | 显示全部楼层 |阅读模式
书目名称Endocrinology of Cardiovascular Function
编辑Ellis R. Levin,Jerry L. Nadler
视频video
丛书名称Endocrine Updates
图书封面Titlebook: Endocrinology of Cardiovascular Function;  Ellis R. Levin,Jerry L. Nadler Book 1998 Springer Science+Business Media New York 1998 Angiotens
描述.Endocrinology of Cardiovascular Function. is a fittinginauguration to the .Endocrine Update Series.. The aim of thesepublications is to provide the clinician with cutting edge, yetsuccinct, access to the latest advances in endocrinology.Historically, our understanding of hormonal disturbances wasrestricted to the classical secretory glands and their targets. As.Endocrinology of Cardiovascular Function. so aptly indicates,endocrinology is no longer constrained by our early physiologicunderstanding of glandular disorder. .Endocrinology of..Cardiovascular Function. has set the standard of excellence forthe future volumes in this series. .Shlomo Melmed, M.D. Series Editor, .Endocrine Update. .Growth factors such as IGF-1 play important roles in cardiovascularcell hypertrophy and the response to acute vascular injury. Fromanother perspective, traditional endocrine hormones, such as estrogen,have been found to participate in preventing the development ofatherosclerosis in women, acting through novel mechanisms on targetvascular cells. Other `endocrine‘ hormones, such as PTHRP andadrenomedullin, also modulate cardiovascular and renovascular dynamicstates, suggesting new roles for these p
出版日期Book 1998
关键词Angiotensin II; Diabetes; cardiovascular; cardiovascular function; estrogen receptor; growth factor; hyper
版次1
doihttps://doi.org/10.1007/978-1-4615-5569-8
isbn_softcover978-1-4613-7547-0
isbn_ebook978-1-4615-5569-8Series ISSN 1566-0729
issn_series 1566-0729
copyrightSpringer Science+Business Media New York 1998
The information of publication is updating

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Sex Steroids and Endothelinprevalence cf cardiovascular disease is significantly less in premenopausal women than inagematched men. There is an approximate 10 year time lag in the development of coronary artery disease in premenopausal women compared to men, however, this gap appears to narrow after the menopause (., .). Such
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Effects of Sex Steroids in Vascular Injurych lower in premenopausal women than in age matched men, but rises steadily in women after the menopause (.). Epidemiologic and observational clinical studies have shown that postmenopausal women on estrogen replacement therapy have less severe coronary artery disease and a lower risk of cardiovascu
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Angiotensin II, Growth Factors and the Lipoxygenase Pathway balloon angioplasty. These processes are mainly mediated by growth factors and cytokines released by various cells in the vessel wall. However, the role of free lipids in these events are not very clear. Vascular smooth muscle cell growth factors and cytokines can activate phospholipases with the r
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AT1 versus AT2 receptorgulating blood presure and hydromineral homeostasis. This octapeptide is generated by cleavage of the aminoterminus of the substrate angiotensinogen, a 60 kDa liver protein belonging to the serine protease inhibitor family, by the aspartyl protease renin, produced by the kidney juxtaglomerular appar
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Physiology of Natriuretic Peptidesa unique 17 amino acid residue ring structure (Fig 1) which largely confers bioactivity and specificity (.). Originally isolated from rat atrial muscle extracts (.), atrial natriuretic peptide (ANP) was the first of the family to be identified and characterised as a circulating hormone responsive to
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Insulin and Insulin-Like Growth Factor I (IGF-I) Effects on Ca2+ and Nitric Oxide in Diabetesof the cardiovascular system, regulate a number of important physiological functions [1-60]. There is also emerging evidence that abnormalities in the actions of these peptides contributed to disease states such as diabetes mellitus. This chapter reviews our current understanding of mechanisms by wh
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Protein Glycation and Diabetic Vascular Disease as retinopathy, nephropathy, neuropathy, and atherosclerotic vascular disease [.]. Despite continuing improvements in the management of diabetic glucose control, these severe and lifethreatening end-organ complications continue to develop in many patients. Both insulin-dependent and non-insulin dep
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