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Titlebook: Effectors of Thermogenesis; Proceedings of a Sym L. Girardier,J. Seydoux Conference proceedings 1978 Springer Basel AG 1978 adaptation.cell

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楼主: ergonomics
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Europäische Distributionssystemeorticosteroid or insulin. Both hormones inhibit synthesis of the enzyme in BAT and increase it in liver. Fatty acid synthetase activity is enhanced by these hormones, but only in BAT. It is suggested that corticosteroids may act via insulin release, since both blood glucose and blood insulin levels
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https://doi.org/10.1007/978-3-662-26502-4te with its final subcellular effect are summarized here. The hormone-mediated regulatory pathway consists of the adenyl cyclase and the protein kinase systems; the former is composed of the receptor and catalytic sites, the latter of regulatory and catalytic subunits. Emphasis is given currently to
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Gestaltung von Texten (Formatieren),g processes (Fig.1). Metabolic heat set free by a “normal” rate of oxidative phosphorylation is sufficient to sustain biological temperatures in homeothermic animals kept under thermoneutral conditions. When the environmental temperature is lowered to a hypothermic level, metabolic heat production i
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https://doi.org/10.1007/978-3-658-14868-3ind to a component on the outer face of the inner membrane. This unique mechanism has been proposed to represent the molecular site of non-shivering thermogenesis in this tissue. Using a photo-affinity analogue of ATP, we have identified the nucleotide binding component as a protein of 32,000 dalton
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IX Konzepte radikaler Moralkritik,s suppress S. and S. of αKGO at 0.6–1.17 mM Mg.: some combinations of it and Mg. increase the respiratory control ratio (RCR). Spermine suppresses S. of αKGO and increases the RCR but suppresses the RCR of SO at high levels. In heat-acclimated (HA) animals in which we have studied only spermine effe
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