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Titlebook: Disorders of the Hand; Volume 2: Hand Recon Ian A. Trail,Andrew N.M. Fleming Book 2015 Springer-Verlag London 2015 Hand.Orthopaedics.Surger

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Carpal Tunnel Syndrome usually simple to diagnose and straightforward to treat [3]. CTS is a clinical diagnosis, requiring assessment by a clinician with sufficient experience to filter out those patients that need investigation of an alternative cause of their symptoms. There is concern that the oversimplification of th
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Ulnar Tunnel Syndromeup. Surgical management addresses the underlying cause of compression. This chapter reviews the clinical presentation of ulnar tunnel syndrome, the relevant patho-anatomy, workup, and longitudinal management.
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https://doi.org/10.1007/978-3-662-13122-0 in peripheral nerve surgery and the establishment of the principle of tension free repair [2] allowed inspired surgeons such as Narakas, Millesi, Allieu, Brunelli, Terzis, Doi, Gu, and others to suggest several new approaches to nerve reconstruction.
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https://doi.org/10.1007/978-3-662-13122-0vascularized bone grafts, flap surgery and tendon transfers. The exquisite functioning of the hand is characterised by mobility and sensibility. Motion is achieved by the contractile action of functioning musculo-tendinous units on mobile joints. When a specific function has been lost due to failure
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https://doi.org/10.1007/978-3-662-13122-0logy particularly within the finger. The reconstruction of the long extensors is straightforward involving either tendon buddying, tendon grafting or tendon transfer with a reasonably predictable result. The reconstruction of the extensor mechanism in the finger is far harder and much less predictab
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https://doi.org/10.1007/978-3-662-13122-0 flexor tendon injuries. Ideally, prompt end-to-end repair follows early diagnosis of an acute flexor tendon laceration. In general, primary repair can be attempted up to 3–6 weeks after zone I–V injuries in uncomplicated cases.
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https://doi.org/10.1007/978-3-642-99103-5 acute injury, or missed diagnosis. The risk of non-union increases the more proximal the fracture, as a result of the pattern of intraosseous blood flow of the scaphoid, as well as with displacement, particularly in cases of additional carpal instability [1, 2].
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