Titlebook: c-Myc Function in Neoplasia; Chi V. Dang,Linda A. Lee Book 1995 Springer-Verlag Berlin Heidelberg 1995 biology.cancer.cell.cell death.gene

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Max Association with Myc,ains of Myc to identify Myn, the murine homolog of Max.. Genes or cDNAs of . in other species, such as Xenopus, zebrafish and chicken, have been cloned as we11.. The genomic structure of the chicken gene suggests that it contains a TATA-less promoter which consists of GC-rich sequences resembling bi

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Book 1995to repair spontaneously occurring mutations leads to genom­ ic instability and could potentially result in the accumulation of car­ cinogenic DNA lesions. Finally, activation of proto-oncogenes, which are normal cellular genes, into oncogenes could accelerate the 11 processes of cell proliferation.

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1080-3645 c DNA lesions. Finally, activation of proto-oncogenes, which are normal cellular genes, into oncogenes could accelerate the 11 processes of cell proliferation. 978-3-662-22683-4978-3-662-22681-0Series ISSN 1080-3645

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Historical Perspectives of , Gene Studies,1910 suggested that certain avian sarcomas could be transmitted by a filterable infectious agent.. Subsequently, isolates of the infectious agents were recognized under the electron microscope as membrane bound particles now known as retroviruses. Five of these retroviruses that have been isolated f

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Properties of the c-Myc Protein,mmands that controls transcriptional initiation, elongation as well as mRNA stability. In addition to this hierarchy of controls, production of the c-Myc polypeptides is also regulated. The predicted size of the c-. encoded polypeptide initiated at the canonical AUG translational start site is 439 a