Titlebook: Current Perspectives in microRNAs (miRNA); Shao-Yao Ying Book 2008 Springer Science+Business Media B.V. 2008 DNA.Microarray.RNA Splicing.S

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Sozialreputation in der Mediengesellschaft,regulation of miRNA expression in chronic myeloid leukemia (CML) depending on BCR-ABL, the characteristic onco-protein of CML. Using microarray analysis (miCHIP) and miRNA-specific quantitative real-time RT-PCR from CML cell lines treated with either imatinib or anti BCR-ABL shRNAs to inhibit its ty

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https://doi.org/10.1007/978-3-531-90727-7on and/or mRNA degradation. However, the miRNA expression profile in vessels and their potential roles in vascular diseases are currently unknown. Therefore, we determined miRNA expression in rat carotid arteries using microarray analysis. Indeed, compared with other tissues, tissue-specific express

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Konzeptioneller Rahmen und Fragestellung,gistic manner to re-establish barrier function by regenerating new skin. The inducible expression and repression of genes represents a key component of this process. MicroRNAs (miRNAs) are powerful regulators of gene expression yet their significance in tissue repair remains largely unknown. Recent

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MicroRNA and Erythroid Differentiation,n conclusion, dynamic changes in miRNA expression occurred during erythroid differentiation, with an overall increase in the levels of miRNAs upon terminal differentiation of erythroid cells. MiR-451 may play a role in promoting erythroid differentiation.

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Role of mir-302 MicroRNA Family in Stem Cell Pluripotency and Renewal,ell pluripotency. For this, we developed a Pol-II-based intronic miRNA expression system to transgenically express the mir-302s in several human epidermal and cancerous cell lines. Surprisingly, these mir-302s-transfected cells, namely miRNA-induced pluripotent stem (mirPS) cells, were shown to not

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MicroRNA in Neuropsychiatric Diseases,hanisms implicated in Parkinson disease; or enhance neurodegeneration in Drosophila model induced by proteins implicated in polyQ neurodegenerative pathologies. Loss of FMPR protein function is a cause of common mental retardation syndrome (FXS) and function of FMRP or FMPR-related protein in . (Dfm