Titlebook: Crossroads between Innate and Adaptive Immunity; Peter D. Katsikis,Stephen P. Schoenberger,Bali Pul Conference proceedings 2007 The Editor

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The Yin and Yang of Adaptive Immunity in Allogeneic Hematopoietic Cell Transplantation: Donor Antigong-term leukemia- free survival with minimal GvHD when supralethal doses of LBRM were administered before transplant, or 75 days after BMT. Higher levels of serum gamma interferon and expansion of spleen-derived CD4. memory T cells were seen among recipients of CD11b-depleted BM compared to recipie

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Innate Tumor Immune Surveillance,e recipient, the pretransplant infusion of donor-vs.-recipient alloreactive NK cells obviated the need for high-intensity conditioning and conditioned the recipients to BMT without GVHD.,.. Several other studies also strongly support a key role for innate cells and mechanisms in controlling tumor in

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synovial-joint

CD28 and Cd27 Costimulation of Cd8+ T Cells: A Story of Survival,nals that involve multiple members of the B7:CD28 and TNFα/TNFR families. This review will focus on CD28 and CD27 costimulation and examine their involvement in the costimulation of CD8+ T cell responses and the role of such costimulation in the survival of activated, resting, and memory CD8+ T cell

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Nausea

Vascular Leukocytes: a Population with Angiogenic and Immunossuppressive Properties Highly Represenrization.,.. Pathological neovascularization is characterized by increased vascular permeability, which leads to leakage, hemorrhaging, and inflammation. Although sprouting of blood vessels is the principal process in neovascularization, other mechanisms such as intussusception or cooptation of circ

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Receptors and Pathways in Innate Antifungal Immunity,hrough antigenic variability, phenotypic switching, and dimorphic transition is the existence of a multitude of recognition and effector mechanisms to oppose fungal infectivity at the different body sites.

epicardium

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nsights into the molecular mechanism of action of YF-17D, and highlight the potential of vaccination strategies that use combinations of different TLR ligands to stimulate polyvalent immune responses.

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